Atentie la capuse,la Borelioza,la Lyme! (4)

wannajo spune:

Inca sunt aici..astept raspuns, desi e noapte..sunt din ce in ce mai disperata, am apucat sa citesc mai multe info aici...

Ma poate indruma cineva si la ce pediatru sa mergem? Sau la ce spital? Ma gandeam la gr. Alexandrescu dar am inteles ca a mai fost cineva si nu a fost ok...
Daca ma puteti ajuta este excelent, vreau sa merg cat mai dimineata.

Multumiri multe!!!

ionescu.cris spune:

Buna ,
Incerc eu sa va raspund la intrebari :
Daca omit ceva in raspunsul meu ,cei cu experienta pot veni cu informatii in plus care pot ajuta .

Alina :
si in cazul nostru a fost un eritem redus la inceput care ulterior a s-a marit avand o dimensiune de 20-30 cm .
Din pacate am confundat-o cu o piscatura de tantari , si simptomele care au aparut dupa cateva zile medicul le-a pus pe seama unei gripa .
In afara de tratam cu doxy nu ai ce face ; doar sa-l asociezi cu Plaquenil .
Am mai scris ca bacteria are un invelis foarte rezistant si antibioticul nu are capacitatea sa-l distruga ca sa ajunga la genom .
Ori acest Plaquenil poate dizolva acest invelis , usurand munca antibioticului , crescandu-i astfel eficacitatea .In locul Plaquenilului se foloseste Artemisinin ( extras din Atemisia annua). Ambele sunt folosite in tratarea malariei , ultimul neavand efecte secundare . In cazul Plaquenilului trebuie facut control oftalmologic .
Deci pentru un tratametn eficace , cele doua produse trebuiesc asociate .

*De regula tratam se aplica pana la disparitia simtpomelor ,si in cazul in care starea de sanatate se reechilibreaza , anual se aplica un tratam de sustinere cu antibiotic astfel mentinandu-se controlul asupra bacteriei .

*De la muscatura de capusa nu poate apare reactie alergica .
Nu uita ca o capusa mai poate transmite alte bacterii si virusi .

Sergiu :
Toti cei care au ajuns la Budapesta erau la capatul puterilor daca pot folosi expresia . Dupa zeci de teste la diferite laboratoare , medici viziti si fara un diagnostic precis ,si acuzand gama de simptome pe care le cred cunosti deja ,la Dr Bozsjik aveau 2 posibilitati : ori sa aiba sifilis ori borrelia .Cum marea mai toti aveau amitirea muscaturii de capusa nu aveau cum sa primeasca un diagnostic negativ .
La multi au fost gasite formele spirochetelor specifice borreliei garinii ,afzelii , combinate cu burgdorferi .
Cred ca subiectul il poti aprofunda cu doctorul , cu atat mai mult cu cat nu isi cronometreaza timpul si ofera cu tot profesionalismul explicatii pe intelesul tuturor .
Dupa test iti va da si un "film " cu ceea ce se "vede" in probele tale de sange .

wannajo
in stadiul actual nu se fac analize .
Dupa opinia Dr Tilea de la Tg Mures se aplica tratament preventiv in urgenta .
Cauta un medic care sa accepte sa prescrie imdiat 10 zile de antibiotic.
In faza asta chiar daca a fost o capusa infestata bacteria sansa de a stopa boala este maxima .
In Bucuresti nu cunosc nici un pediatru sau medic care sa se ocupe de copii .

Dragoslav care a scris anterior ar putea sa dea informatii mai ample .

Va doresc multa sanatate
Cris

aalina2003 spune:

Am si eu o intrebare pentru voi. Matusa mea, 50 ani, din Constanta, a fost dignosticata cu scleroza laterala amiotrofica. Boala este groaznica,letala, in 3-6 ani de la debut. Debutul se pare ca a vut loc acum 6 luni,diagnosticul a fost pus de-abia acum, o vreme a fost suspecta de miastenia gravis.In fine,tot rasfoind, am vazut ca simptomele pot fi date si de Lyme. Vrem sa aflam daca este asa,chiar daca este tarziu.
Care sunt pasii de urmat?
1.Teste de sange: care sunt concludente si unde se pot face? (Bucurestiu sau Constanta).Inteleg din postarile voastre ca nu oriunde si nu orice metoda de analiza este concludenta. CEva nume de laboratoare,nr de tel?
2. daca sunt negative,poate fi totusi lyme? trebuie sa ajunga la Tg mures la dr Butiriuca, la Budapesta, la dr Botzig? Numere de telefon?
Va rog mult,ajutati-ma sa gasesc raspunsul la intrebarea mea.

Mariaa spune:

daca RMN-ul indica scleroza multipla, atunci nu mai e nici un dubiu!
Lyme da simptome de scleroza multipla insa aceasta poate fi infirmata la RMN.

daca insa Lyme nu este depistata la timp si nu s-a intrat pe tratament corespunzator, in timp poate provoca daune la nivel de sistem nervos cum ar fi demielinizarile specifice sclerozei multiple.

pe de alta parte varsta matusii tale este atipica pentru declansarea SM. cat de avansate sunt simptomele? ce se vede la RMN-ul matusii tale?

_

“Before you speak, listen. Before you write, think. Before you spend, earn. Before you invest, investigate. Before you criticize, wait. Before you pray, forgive. Before you quit, try. Before you retire, save. Before you die, give.”
Emma este IUBIRE ****** un copil creste in inima mea ****** am un inger pazitor

aalina2003 spune:

Diagnosticul este scleroza laterala amiotrofica. Exista diferente fata de scleroza multipla,dar nu stiu exact in ce constau.
Debutul bolii a fost atipic,adica tulburari de vorbire (pronuntie) si de inghitire. Tipic ar fi fost sa apara intai tulburari de motricitate, abia catre final cele de vorbire. Acum..i se pare ca a simtit ceva slabiciune musculara in urma cu 10 ani in urma,darnu stiu daca pot pune baza in afirmatia asta pentru ca:
-testul EMG a iesit normal,(care tine de o buna functionare motrica)de aici s-a tras concluzia ca nu ar fi SLA
-a mai facut un test la nivelul limbii care a fost relevant pentru diagnosticul de SLA
-la un RMN fara substanta de contrast nu s-a vazut nimic. A repetat (cu substanta de contrast) si au aparut leziuni de trunchi cerebral, care ar fi putut tine si de un infarct de trunchi cerebral nu neaprat de SLA
Oricum,problema este la nivelul trunchiului cerebral, de asta nu poate vorbi, dar ...poate,totusi,nu e SLA.

ionescu.cris spune:

Un material care intereseaza pe toti care acceseaza topicul Lyme :

300 MEDICAL CONDITIONS
RELATED TO LYME BORRELIOSIS

Frequently Misdiagnosed

Katrina Tang, M.D., HMD, founder and Director of Research at
the Sierra Integrative Medicine Clinic in Reno, Nevada, states
that Lyme disease eludes many doctors because of its ability
to mimic many other diseases. According to an informal
study conducted by the American Lyme disease Alliance
(ALDA), most patients diagnosed with Chronic Fatigue
Syndrome (CFS) are actually suffering from Lyme disease.

Dr. Paul Fink, past president of the American Psychiatric Association, has acknowledged that Lyme disease can contribute to every psychiatric disorder in the Diagnostic Symptoms Manual IV (DSM-IV).
This manual is used to diagnose psychiatric conditions such as attention deficit disorder (ADD), antisocial personality, panic attacks,anorexia nervosa, autism and Aspergers syndrome (a form of autism) to name a few.

List of Conditions
Lyme Borreliosis causes, mimics, is manifested as, is misdiagnosed as or is a contributing factor to many conditions. The following list of over 300 conditions was compiled by means of a non exhaustive search of published scientific literature and
includes:

Abdominal pseudo-eventration,
Acrodermatitis chronica atrophicans (ACA)
Acute Acral Ischemia
Acute conduction disorders
Acute coronary syndrome
Acute exogenous psychosis
Acute meningitis
Acute myelo-meningo-radiculitis
Acute peripheral facial palsy
Acute perimyocarditis
Acute pyogenic arthritis
Acute reversible diffuse conduction system disease
acute transitory auriculoventricular block
Acute transverse myelitis
Acute urinary retention
Acquired Immune Deficiency Syndrome (AIDS)
Algodystrophy
Allergic conditions
Allergic conjunctivitis
Alopecia
Alzheimer’s Disease
Amyotrophic lateral sclerosis
(ALS - Lou Gehrig’s Disease)
Amyotrophy
Anamnesis
Anetoderma
Anorexia nervosa
Antepartum fever
Anxiety
Arrhythmia
Arthralgia
Arthritis
Asymmetrical hearing loss
Atraumatic spontaneous hemarthrosis
Atrioventricular block
Attention Deficit Disorder (ADD)
Attention Deficit Hyperactivity Disorder
(ADHD)
Bannwarth’s Syndrome
Behcet’s disease
Bell’s Palsy
Benign cutaneous lymphocytoma
Benign lymphocytic infiltration (Jessner-Kanof)
Bilateral carpal tunnel syndrome
Bilateral facial nerve palsy
Bilateral follicular conjunctivitis
Bilateral keratitis
Bilateral papilloedema
Biphasic meningoencephalitis
Bipolar Disorder
Brain Tumor
Brown recluse spider bite
Brown-Sequard syndrome
Cardiac Disease
Cardiomegaly
Cardiomyopathy
Carditis
Carpal tunnel syndrome
Catatonic syndrome
Cauda equina syndrome
Central vestibular syndrome
Cerebellitis
Cerebral atrophy
Cerebro-vascular disease
Cervical facet syndrome
Cheilitis granulomatosa
Chiasmal optic neuritis
Chorea
Choriocapillaritis
Chronic encephalomyelitis
Chronic Fatigue Syndrome
Chronic muscle weakness
Chronic urticaria
Cerebellar ataxia
Cogan’s syndrome
Collagenosis
Complete flaccid paraplegia
Complex Regional Pain Syndrome (CRPS)
Concomitant neuroretinitis
Conduction disorder
Conus medullaris syndrome
Coronary aneurysm
Cortical blindness
Coxitis
Cranial Neuritis
Cranial polyneuritis
Craniopharyngioma
Cutaneous B-cell lymphoma
Dementia
Demyelinating disorders
Depression
Dermatomyositis
Diaphragmatic paralysis
Diffuse fasciitis
Dilated cardiomyopathy
Diplopia
Discopathy
Disseminated choroiditis
Dorsal epiduritis
Encephalitis
Encephalomyelitis
Encephalopathy
Endogenous paranoid-hallucinatory syndrome
Eosinophilia
Eosinophilic fasciitis (Shulman syndrome)
Epilepsy
Epileptic crises
Episcleritis
Epstein Barr
Erythema chronicum migrans
Exanthema (local and generalized)
Extrapyramidal disorders
Facial diplegia
Fascicular tachycardia
Fatal adult respiratory distress syndrome
Fetal death
Fever
Fibromyalgia
Fibrositis
Focal nodular myositis
Frontotemporal atrophy
Generalised motor neuron disease
Geniculate neuralgia
Giant cell arteritis
Gonarthritis
Granuloma annulare
Guillain-Barré Syndrome
HLA-B27 negative sacroiliitis
Headaches (severe)
Hearing loss
Heart block
Hemiparesis
Hemophagocytic syndrome
Hepatic disorders
Hepatitis
Herniated discs
Holmes-Adie syndrome
Horner’s syndrome
Human necrotizing splenitis
Hydrocephalus
Hyperacusis
Hyperbilirubinemia
Hypothyroidism
Idiopathic atrophoderma of Pasini and Pierini
(IAPP)
Idiopathic facial paralysis
Infarction pain
Impaired Brainstem response
Infantile sclero-atrophic lichen
Infectious Mononucleosis
Infiltrating lymphadenosis benigna cutis
Inflammatory cerebrospinal fluid syndrome
Influenza
Internuclear ophthalmoplegia
Interstitial granulomatous dermatitis
Intracerebral haemorrhage
Intracranial aneurysm
Intracranial hypertension
Intracranial mass lesions
Intrauterine growth retardation
Iritis
Irritable Bowel Syndrome
Isolated acute myocarditis
Isolated lymphadenopathy
Isolated neuritis of the sciatic nerve
Isolated oculomotor nerve paralysis
Isolated posterior cord syndrome
Jaundice
Juvenile Rheumatoid Arthritis
Keratitis
Keratoconus
Left sided sudden hemiparesis
Lichen sclerosus
Livedo racemosa
Lofgren’s syndrome
Lupus
Lymphadenosis benigna cutis
Lymphocytoma cutis
Lymphoma
Lumboradicular syndrome
Melkersson-Rosenthal syndrome
Memory impairment
Meningeal lymphoma
Meningitis
Meningoencephalomyelitis,
Meningoencephalomyeloradiculoneuritis
Meningoradiculitis
Migraines
Mono-arthritis
Monolateral chorioretinitis
Morgagni-Adams-Stokes syndrome (MAS)
Morning glory syndrome
Morphea
Motor neuron syndrome
Multiple mononeuropathy
Multiple Sclerosis
Myelopathy
Myofascial pain syndrome
Myositis
Neonatal respiratory distress
Neuromyotonia
Nodular panniculitis
Normal-pressure hydrocephalus (NPH)
Oculomotor paralysis
Oligoarthritis
Opsoclonus-myoclonus syndrome
Nodular fasciitis
Non-Hodgkin’s lymphoma
Obsessive-compulsive disorder
Optic atrophy
Optic disk edema
Organic mood syndrome
Optic nerve lesion
Otoneurological Disorders
Panuveitis
Papillitis
Paralysis of abdominal muscles
Paraneoplastic polyneuropathyLumboradicular syndrome
Melkersson-Rosenthal syndrome
Memory impairment
Meningeal lymphoma
Meningitis
Meningoencephalomyelitis,
Meningoencephalomyeloradiculoneuritis
Meningoradiculitis
Migraines
Mono-arthritis
Monolateral chorioretinitis
Morgagni-Adams-Stokes syndrome (MAS)
Morning glory syndrome
Morphea
Motor neuron syndrome
Multiple mononeuropathy
Multiple Sclerosis
Myelopathy
Myofascial pain syndrome
Myositis
Neonatal respiratory distress
Neuromyotonia
Nodular panniculitis
Normal-pressure hydrocephalus (NPH)
Oculomotor paralysis
Oligoarthritis
Opsoclonus-myoclonus syndrome
Nodular fasciitis
Non-Hodgkin’s lymphoma
Obsessive-compulsive disorder
Optic atrophy
Optic disk edema
Organic mood syndrome
Optic nerve lesion
Otoneurological Disorders
Panuveitis
Papillitis
Paralysis of abdominal muscles
Paraneoplastic polyneuropathy
Paranoia
Parkinsonism
Parotitis
Pars plana vitrectopy
Parsonage and Turner syndrome
Peripheral facial palsy
Peripheral neuropathy
Peripheral vascular disorder
Pericarditis
Perimyocarditis
Persistent atrioventricular block
Pigment epitheliitis
Polymyalgia rheumatica
Polyneuritis cranialis
Polyneuropathy
Polysymptomatic autoimmune disorder
Porphyrinuria
Posterior scleritis
Primary lymphoma of the nervous system
Presenile dementia
Progressive cerebral infarction
Progressive facial hemiatrophy (Parry-Romberg
syndrome)
Progressive stroke
Progressive supranuclear paralysis
Prolonged pyrexia
Propriospinal myoclonus
Pseudo tumor Cerebrae
Pseudolymphoma
Pseudoneoplastic weight loss
Psychosomatic disorders
Radiculoneuritis
Ramsay Hunt syndrome (pleocytosis)
Raynaud’s syndrome
Recurrent paralysis
Reflex sympathetic dystrophy
Reiter’s Syndrome
Respiratory failure
Restless legs syndrome
Retinal pigment epithelium detachment
Retinal vasculitis
Reversible dementia
Rheumatic Fever
Rheumatoid Arthritis
Rhombencephalitis
Sacro-iliitis infection
SAPHO syndrome
Sarcoidosis
Schizophrenia
Schoenlein-Henoch purpura
Scleroderma
Secondary syphilis
Seizure Disorders
Sensorineural Hearing Loss
Septal panniculitis
Septic arthritis
Seventh nerve paralysis
Sick sinus syndrome
Spontaneous brain hemorrhage
Stevens-Johnson syndrome
Stiff-man syndrome
Still’s disease
Stroke
Subacute Bacterial Endocarditis
Subacute multiple-site osteomyelitis
Subacute organic psychosyndrome
Subacute multiple-site osteomyelitis
Subacute presenile dementia
Subarachnoid hemorrhage
Sudden deafness
Sudden hemiparesis
Sudden infant death syndrome (SIDS)
Sudeck’s atrophy
Synovitis
Syphilis
Symmetric Polyarthritis
Temporal arteritis
Temporomandibular joint syndrome
Thrombocytopenic purpura
Thyroiditis
Tourette’s syndrome
Transient Ischemic Attack
Transient left ventricular dysfunction
Trigeminal Neuralgia
Unilateral interstitial keratitis
Unilateral papillitis
Urticaria
Uveitis
Vasculitic neuropathy
Vasculitic mononeuritis multiplex
Vasculitis
Ventricular asystole
Vertigo
Vestibular neuronitis
Vitreous clouding

EXAMPLES OF MISDIAGNOSIS

The following stories of 4 individuals with diagnosis of ALS illustrate how important early diagnosis is.

THE FIRST is an individual with a 10-year diagnosis of ALS from whom we received a spinal fluid and blood specimen. The spinal fluid was highly positive for Bb, as was the blood. We reported the findings within a 24-hour period of receiving the specimens only to learn that the individual had died.

THE SECOND individual also had a long history of problems identified as ALS. His RIBb test was positive and he was not able to get any physician to treat him for Lyme disease. His health deteriorated and he was admitted to a hospital and was on life support. When his wife was told of his impending death she obtained a court order to have him treated with antibiotic therapy for Lyme disease. He recovered enough to get off life support and was subsequently discharged. He gained weight (32 pounds) and lived eight more months and then died of a heart attack.

THE THIRD individual is a 25-year-old golfer on a golf team. He became very ill and was unable to play golf. He was diagnosed with ALS. A family friend knew about our test and sent a blood specimen to be tested for Lyme disease. The results were positive. He was started on appropriate antibiotic therapy and was soon able to resume his golf career. He is now a professional golfer. Having an early diagnosis seems to have made the difference for this young man in living a productive active life.

THE FOURTH is a young college student who began having cognitive difficulties and had to drop out of school. He learned about our laboratory and was tested and found to be positive for Lyme disease. After four months on antibiotic he was able to resume his normal active life and is on the deans list and writing classical music.

These examples may shed some light on the importance of early diagnosis and appropriate treatment for Lyme disease. Left untreated
the out-come of Lyme disease can result in a chronic debilitating condition and possible death. Are you sure you don’t have Lyme
disease?

Dr. Jo Anne Whitaker is President and Director of Research at Bowen Research & Training Institute, Inc. 38541 US Highway 19 North,
Palm Harbor, Florida .
Tel: 727-937-9077
Email: JoAnne@bowen.org
Web: www.bowen.org
Sursa :
NutraNews / New Thinking, New Discoveries in Nutraceutical Research

wannajo spune:

Buna seara,
multe multumiri ionescu.cris!
Am fost azi la matei bals, la contagioase la infectionist. Ne-a prescris Augmentin ES 5,5 ml la 12 ore, timp de 10 zile. Asa ne zisese de dim si pediatra. Deasemenea ne-a spus sa revenim pt recoltare de sange dupa 2 saptamani. Nu ne-a spus si pt ce anume se vor recolta probele...
Deasemenea ne-a trimis la Gr Alexandrescu la chirurgie pt un controlamanuntit, unde ne-a spus d-na dr ca cei de acolo se vor uita cu lupa speciala si ne vor spune daca nu cumva a ramas ceva sub piele.

La Alexandrescu dezamagire totala (NU VA RECOMAND SA MERGETI ACOLO CU CEI MICI PT CAZURILE ACESTEA). O d-na dr plictisita s-a uitat la locul muscaturii cu ochii, fara lupa sau altceva, a zis ca nu e nici un piciorus acolo, a dat cu iod si ceva alb praf (parea antibiotic) si ne-a spus ca asistenta ii va face vaccinul antitetanos imediat. Daca nu aveam la mine carnetelul de vaccinari si nu stiam ca in schema de vaccinare este inclus antitetanosul ni-l facea fara intrebari sau cererea acordului...In plus ne-a zis ca nu ne trebuie nici un antibiotic, doar comprese cu rivanol (nu i-am pus, nu stiu daca e bine) si maine sa revenim sa il vada pe copil.
Asadar nu mai merg maine tot acolo...

Am inceput cu augmentinul ES, deja i-am dat prima doza la ora 21.
Ce parere aveti, tratamentul este cel corect, este toxic acest tip de antibiotic? Ni s-a spus ca augmentinul ES merge pe borellia dar nu mi-a spus nimic despre lyme sau altceva.
Ce stiti despre asta? Si o fi suficient 10 zile?
Apoi ce analize se vor recolta, voi avea o certitudine in urma primirii rezultatului?
Se fac teste numi pt borellia sau si pt lyme si altele?
trebuiesc repetate analizele periodic?
Deasemenea, ce trebuie sa urmaresc pana atunci, timp de 2 saptamani?

Imi cer scuze de multele nedumeriri si va multumesc mult pt ajutor!
Sanatate si mult noroc tuturor!!!

alinamoro spune:

Cris, multumesc foarte mult pentru raspuns.
O sa caut Artemisinin, daca nu gasesc aici caut in Ungaria, eventual il intreb si pe medicul homeopat.

---

Alina cu www.snugglepie.com/ezb/653339.png" target="_blank"> Adelina (31.08.2006) & www.snugglepie.com/ezb/653351.png" target="_blank">Dragos (11.04.2002) poze

cataset spune:

Am discutat cu Dr. Bozsik in legatura cu copilul de 2 ani jumate muscat de capusa acum 6 saptamani si mi-a scris ca trebuie sa ii dau cel putin 25 zile antibiotic ca sa fiu sigur .

Reguli generale: daca la inceputul tratamentului apar manifestari de reactie la medicament inseamna ca antibioticul este eficient . Cea mai mare greseala este întreruperea precoce a tratamentului antibiotic! Consumul de lichide permanent si în cantitatii mari ajuta; la eliminarea substantelor descompuse care rezulta în urma distrugerii bacteriilor. Înrautatirea tranzitorie a starii pacientului se datoreaza efectului medicamentelor, deci este un fenomen favorabil. Începând cu ultima zi de tratament antibiotic este foarte important consumul de cantitati mari de iaurt natural, cu microflora vie, respectiv preparate continand Lactobacili. Se recomanda ca medicamentele sa fie luate în timpul meselor, împartind proportional dozele zilnice! Avand rezultate pozitive de IgM si rezultate negative de IgG la aprox 2 luni indica o infectie recenta si tratabila usor. Reactia JHR in prima perioada a tratamentului inseamna ca antibioticul este eficient , recomandare min 25 zile. In timpul tratamentului cu antibiotic iaurturile nu sunt eficiente ele se administraza dupa terminarea lor pentru refacerea florei intest si a imunitatii.

ionescu.cris spune:

Wannajo
Incepe tratamentul si ar fi bine sa procedezi la fel ca si Cataset : Sa contactezi pe Dr Boszik sau Dna Dr Tilea la Tg Mures .
Augmentinul este folosit in tratament in cazul copiilor .
Borrelioza sau Lyme desemneaza aceeasi boala ;
In rezultatele testelor ... din pacate nu poti avea incredere .. este o realitate , si nici un doctor nu poate contesta aceasta afirmatie .
In tot acest timp si dupa trebuie sa urmaresti evolutia starii de sanatate a copilului .
Nu cred ca superi pe nimeni punand intrebari , ingrijorarea ta este justificata .
Incerca sa gasesti Samento .
Am aratat de fiecare data ca este un produs foarte , foarte bun in tratamentul bolii .

M-am gandit la Dragoslav care afirma ca are o firma pentru produse de acest gen .

Cred ca ar putea lua legatura cu Nutramedix si ar aduce Samento in tara , din momentul in care se gaseste in Bulgaria si Ungaria de ce nu ar fi si pe piata in Romania ??

AALINA 2003
In legatura cu matusa ta puteti face testele tot la Budapesta .
Poate merge o alta persoana cu sangele ; luati legatura cu doctorul .

Va doresc numai bine
Cris